Toxic plaque buildup in the brain may play a role in the disease, but in the search for effective treatments, researchers are looking to other potential causes.
and a series of failed clinical trials over the years targeting amyloid have left some in the field demoralized.in June that its amyloid targeting drug, crenezumab, failed to slow or prevent cognitive decline in people with a rare genetic mutation that causes early-onset Alzheimer’s disease. The phase 3 trial, which the National Institute on Aging supported, enrolled around 250 people.
“These blood markers are being deployed widely in research studies now and there’s a fair amount of optimism that in the coming years, they will be more widely deployed in the clinic,” Ramanan said.Since new pharmaceutical treatments may be years from being available for patients, some Alzheimer’s researchers are looking more to early detection and prevention such as exercise to slow the onset or progression of the disease.
The data suggest exercise"may be a mechanism of potentially reducing risk for not only developing dementia" but"an overall healthy, balanced lifestyle approach to risk reduction," said Edelmayer, of the Alzheimer’s Association.
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