The study published in Nature Cardiovascular Research reveals that SARS-CoV-2, the virus causing COVID-19, can directly infect coronary vessels and arterial plaques, heightening the risk of cardiovascular complications like heart attacks and strokes. These findings offer critical insights into the increased long-term cardiovascular risks for COVID-19 survivors and open new avenues for targeted therapies.
By Vijay Kumar MalesuOct 1 2023Reviewed by Susha Cheriyedath, M.Sc. In a recent study published in the journal Nature Cardiovascular Research, a group of researchers determined whether Severe Acute Respiratory Syndrome Coronavirus 2 directly infects coronary vessels and atherosclerotic plaques to understand its role in inducing plaque inflammation.
Unraveling COVID’s Heart Connection The present study strictly conformed to ethical standards, examining autopsy specimens from eight COVID-19 patients to investigate the presence of SARS-CoV-2 ribonucleic acid in coronary macrophages, utilizing advanced methodologies such as RNAscope in situ hybridization and spatial artificial intelligence .
Why COVID-19 is a Game-Changer for Heart Health The study uncovered pivotal data about the susceptibilities of vascular smooth muscle cells and macrophages to SARS-CoV-2, showing a higher vulnerability in macrophages. The research disclosed that both macrophages and foam cells, which are associated with atherosclerosis, could host the virus. Notably, foam cells demonstrated more susceptibility and a slower virus clearance process.
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