Exceeding weekly alcohol recommendations has been linked to shorter telomeres on the end of chromosomes, which have been associated with Alzheimer's disease, cancer and heart disease
that found 93 genetic variants associated with increased alcohol use.
This method, called mendelian randomisation, groups people by the genetic variants they possess that have been linked to particular behaviours. The idea is these genes were randomly allocated at conception and are therefore not affected by lifestyle factors. In both the Biobank and GWAS experiments, the team found that the participants who had been diagnosed with alcohol use disorder had the shortest telomeres. But shorter telomeres were also seen in those who were genetically more likely to consume between 17 and 28 units a week.
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